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In the end I learnt why  my penis never developed  as I hit puberty . Here in my country they don't do circumcision to baby so I was safe from that botchery . As I became more self aware I felt naked like Adam and Eve . Hiding the deformity of my body to the naked eye was impossible . My right of freedom and respect stripped away as I became outcassted by everyone . Getting by each day became more difficult . Earning a living a  pain in the ass to put food on the table . Truth be told  I'm just waiting for my time to run out or someone to end my life ! 

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6 hours ago, myfault said:

In the end I learnt why  my penis never developed  as I hit puberty . Here in my country they don't do circumcision to baby so I was safe from that botchery . As I became more self aware I felt naked like Adam and Eve . Hiding the deformity of my body to the naked eye was impossible . My right of freedom and respect stripped away as I became outcassted by everyone . Getting by each day became more difficult . Earning a living a  pain in the ass to put food on the table . Truth be told  I'm just waiting for my time to run out or someone to end my life ! 

I'm having difficulty understanding your posts.

I have no clue what you mean when you say, "In the end I learnt why my penis never developed as I hit puberty."

Why do you think it didn't develop?  I'd be very interested to know! 

Mine developed only very little when my very late and slow puberty finally occured.  My puberty was absurdly slow occured from 16-25 years of age!

Are you saying your lack of development had something to do with circumcision?

Why was your penis so obvious to everyone, if that is what you are saying.  Mandatory nudity for showering around athletics was a grave problem for me, but most of the time my penis wasn't obvious.

Just trying to understand.   Oh, and why "My Fault"?  I have no clue what might have been "your fault."

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On 2/2/2020 at 1:46 PM, uptight outasight said:

I'm having difficulty understanding your posts.

This might perhaps help a bit: http://www.mentalsupportcommunity.net/topic/10515-ruined-my-life-as-a-child/

 

On 2/2/2020 at 7:32 AM, myfault said:

Getting by each day became more difficult . Earning a living a  pain in the ass to put food on the table . Truth be told  I'm just waiting for my time to run out or someone to end my life ! 

I'm sorry you've been struggling and suffering like this :( . 

How does your life get more and more difficult?

Also, why is earning a living so difficult? Do you have a horrible, demanding, exhausting job?

Is there someone you could talk to, about your problems?

In any case, we're here to listen...

Take care!

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  • 1 month later...
On 2/2/2020 at 1:46 PM, uptight outasight said:

I'm having difficulty understanding your posts.

I have no clue what you mean when you say, "In the end I learnt why my penis never developed as I hit puberty."

Why do you think it didn't develop?  I'd be very interested to know! 

Mine developed only very little when my very late and slow puberty finally occured.  My puberty was absurdly slow occured from 16-25 years of age!

Are you saying your lack of development had something to do with circumcision?

Why was your penis so obvious to everyone, if that is what you are saying.  Mandatory nudity for showering around athletics was a grave problem for me, but most of the time my penis wasn't obvious.

Just trying to understand.   Oh, and why "My Fault"?  I have no clue what might have been "your fault."

Did you get your Testosterone, LH and FSH checked at some point? 

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On 3/10/2020 at 2:45 AM, Reality said:

Did you get your Testosterone, LH and FSH checked at some point? 

Do LH and FSH refer to:  Luteinizing and Follicle Stimulating Hormones?

I've read a little about that, but I was never tested for that nor was it even discussed by either of the 2 doctors who prescribed testosterone for me.

They prescribed based on total testosterone on a scale from approx 200 to 900.  My readings were just below the 200 level, so I qualified.  They both thought my levels were declining due to age, but given my history, I think I was always very low.  One also measured free T in the blood, but I can't recall the detail.  It was low.

Testosterone supplementation drastically improved my mental state even given I was over 50 years old.

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2 hours ago, uptight outasight said:

Do LH and FSH refer to:  Luteinizing and Follicle Stimulating Hormones?

I've read a little about that, but I was never tested for that nor was it even discussed by either of the 2 doctors who prescribed testosterone for me.

They prescribed based on total testosterone on a scale from approx 200 to 900.  My readings were just below the 200 level, so I qualified.  They both thought my levels were declining due to age, but given my history, I think I was always very low.  One also measured free T in the blood, but I can't recall the detail.  It was low.

Testosterone supplementation drastically improved my mental state even given I was over 50 years old.

Unfortunately, as long as you receive Testosterone, it won't make a difference. If your gonadotropin (LH and FSH) levels would be too low there would be many things you could do. Clomiphene, Tamoxiphene, Enclomiphene, hCg (LH supplement), FSH injections (e.g. puregon) etc. 

These drugs make it possible to raise testosterone whilst not lowering the volume of the testicles and preserving fertility. Unfortunately, this stuff was rejected by the governments without any rational reason until the research companies went either bancrupt or the patents expired so that research isn't profitable anymore. Since young hypogonadism is very rare, this probably won't change because almost noone cares or even knows about what happened. 

Long-term hCg is not adviced due to "limited data". We found that stuff almost 100 years ago. It's a joke, it's just not funny.

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3 hours ago, Reality said:

Since young hypogonadism is very rare, this probably won't change because almost noone cares or even knows about what happened.

Please comment on this http://www.mentalsupportcommunity.net/topic/9554-question/?do=findComment&comment=106910 and walk us through the process of prenatal and pubescent virilization and the inhibitory roles of endocrine disruptors and environmental xanoestrogens. Also take into account the contemporary "alt-right" meme culture of "soy boys", the politicization of testosterone, avoidance of plastics, etc. 

 

Quote

Low

Undervirilization can occur if a genetic male cannot produce enough androgen or the body tissues cannot respond to it. Extreme undervirilization occurs when no significant androgen hormones can be produced or the body is completely insensitive to androgens. Both result in a female body. Partial undervirilization produces ambiguous genitalia part-way between male and female. The mildest degree of undervirilization may be a slightly small penis. Examples of undervirilization are androgen insensitivity syndrome, 5 alpha reductase deficiency, and some forms of congenital adrenal hyperplasia.

https://en.wikipedia.org/wiki/Virilization

Quote

  Partial androgen insensitivity syndrome (PAIS) is a condition that results in the partial inability of the cell to respond to androgens.[1][2][3] It is an X linked recessive condition. The partial unresponsiveness of the cell to the presence of androgenic hormones impairs the masculinization of male genitalia in the developing fetus, as well as the development of male secondary sexual characteristics at puberty, but does not significantly impair female genital or sexual development.[3][4] As such, the insensitivity to androgens is clinically significant only when it occurs in genetic males (i.e. individuals with a Y chromosome, or more specifically, an SRY gene).[1]

PAIS is one of three types of androgen insensitivity syndrome, which is divided into three categories that are differentiated by the degree of genital masculinization: complete androgen insensitivity syndrome (CAIS) is indicated when the external genitalia is that of a normal female, mild androgen insensitivity syndrome (MAIS) is indicated when the external genitalia is that of a normal male, and partial androgen insensitivity syndrome (PAIS) is indicated when the external genitalia is partially, but not fully masculinized.[1][2][5][6][7][8][9][10][11]

Pubertal undervirilization is common, including gynecomastia, decreased secondary terminal hair, and / or a high pitched voice.[19]The phallic structure ranges from a penis with varying degrees of diminished size and hypospadias to a slightly enlarged clitoris.[1][2][3]Wolffian structures (the epididymides, vasa deferentia, and seminal vesicles) are typically partially or fully developed.[2] The prostate is typically small or impalpable.[20][21]

https://en.wikipedia.org/wiki/Partial_androgen_insensitivity_syndrome

 

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For those of you who think all this shit would go away if only the "liberals" and commies were out of the picture:

https://www.westernmastery.com/2018/02/01/mans-guide-avoiding-xenoestrogens-endocrine-disruptors-lower-testosterone/

Quote

Western Mastery strengthens and educates men amidst the cultural and political chaos that has become the Western world. We push the lifestyle of being a man who is balanced and who always seeks knowledge and development in real-world skills and masculine hobbies. We also educate on real-world issues that affect men.

Social Justice Warriors, far-left liberalism and general degeneracy are not welcome here. This site is for real men who want to change their lifestyle for the better in a culture poisoned by Cultural Marxism.

 

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4 hours ago, uptight outasight said:

They covertly measure by stretching the flaccid as part of their examination.

A much more reliable biological marker than stretching:

https://en.wikipedia.org/wiki/Digit_ratio

Quote

The digit ratios are sexually dimorphic; i.e., most digit ratios are lower in men than in women.

A number of studies have shown a correlation between the 2D:4D digit ratio and various physical and behavioral traits.[23]

Males with CAH have more smaller (more masculine) digit ratios than control males,[7][8] suggesting that prenatal androgens affect digit ratios. Amniocentesis samples show that prenatal levels of testosterone are in the high normal range in males with CAH, while levels of the weaker androgen androstenedione are several fold higher than in control males.[36][37][38]These measures indicate that males with CAH are exposed to greater prenatal concentrations of total androgens than are control males.

A greater digit ratio occurs for men with Klinefelter's syndrome, who have reduced testosterone secretion throughout life compared to control males, than in their fathers or control males.[10]

Correlation with traits

Some authors suggest that digit ratio correlates with health, behavior, and even sexuality in later life. Below is a non-exhaustive list of some traits that have been either demonstrated or suggested to correlate with either high or low digit ratio.

                              Low digit ratio

                                                                                                                  High digit ratio

Physiology and disease
  • Increased risk of prostate cancer and prostate diseases in males.[63][64]
  • Slower utero fetal development in both sexes.[63]
  • Increased reproductive success in males.[54]
  • Longer penis in males.[65]

 

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And also this [WARNING: EXPLICIT PHOTO ON WIKI PAGE]:

https://en.wikipedia.org/wiki/Anogenital_distance

Quote

Studies show that the human perineum is twice as long in males as in females,[2] but males have more variance. Measuring the anogenital distance in neonatal humans has been suggested as a noninvasive method to determine male feminisation and thereby predict neonatal and adult reproductive disorders.[3]

A study by Swan et al. determined that the AGD is linked to fertility in males, and penis size.[4]

Males with a short AGD (lower than the median around 52 mm (2 in)) have seven times the chance of being sub-fertile as those with a longer AGD. It is linked to both semen volume and sperm count.[5] A lower than median AGD also increases the likelihood of undescended testes, and lowered sperm counts and testicular tumors in adulthood. Babies with high total exposure to phthalates were ninety times more likely to have a short AGD, despite not every type of the nine phthalates tested being correlated with shorter AGD.[4]

Swan et al. report that the levels of phthalates associated with significant AGD reductions are found in approximately one-quarter of Americans tested by the Centers for Disease Control and Prevention (CDC) for phthalate body burdens.[4]

Women who had high levels of phthalates in their urine during pregnancy gave birth to sons who were ten times more likely to have shorter than expected AGDs.[6]

 

Biological determinism in action! Tough to get any more "concrete" than that!

 

Mirror, mirror on the wall,

Who's the biggest dick of all?

Cop a squat, grab that tape,

just how long is your 'taint? LOLOLOL

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5 hours ago, Victimorthecrime said:

@Reality I take it you are in the medical field?  What advice would you give guys worried all these various issues?  Eat right, exercise, get sleep, easy on the booze?  Anything else?

Well yes, but actually no. I studied it for 2 semesters and I'm still enrolled but i currently don't plan on finishing it. Nonetheless, I still research stuff I'm interested in and you can get the exact same information a medical professional gets in your local library or on the internet. 

I don't have a way to make your thing bigger by default. Getting harder errections might help (cock rings, viagra, cialis, testo etc.). Flaccid size might decrease with higher blood pressure, so I would try to keep that down - as should anybody anyway, so measure your blood pressure. 

Trimming your body hair should be obvious, but don't shave it clean if you're too small because it makes you look too much like a child. 

Penis pumps might make your penis bigger temporarily. I'm not sure about long-term effects atm.

If you're still in puberty, testosterone (DHT to be precise) might help. Other stuff that might help with development is LH, FSH, IGF-1, GnRH and Kisspeptin. Nonetheless, getting anything but Testo will be hard to impossible.

Other than that there's not much you can do atm to make you penis (look) bigger, I guess.

 

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4 hours ago, Klingsor said:

Please comment on this http://www.mentalsupportcommunity.net/topic/9554-question/?do=findComment&comment=106910 and walk us through the process of prenatal and pubescent virilization and the inhibitory roles of endocrine disruptors and environmental xanoestrogens. Also take into account the contemporary "alt-right" meme culture of "soy boys", the politicization of testosterone, avoidance of plastics, etc. 

 

 

What does that have to do with my quote? 

Anyway, I never researched that topic so I can't comment on it, sorry. 

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On 3/11/2020 at 9:31 AM, Reality said:

Unfortunately, as long as you receive Testosterone, it won't make a difference. If your gonadotropin (LH and FSH) levels would be too low there would be many things you could do. Clomiphene, Tamoxiphene, Enclomiphene, hCg (LH supplement), FSH injections (e.g. puregon) etc. 

These drugs make it possible to raise testosterone whilst not lowering the volume of the testicles and preserving fertility. Unfortunately, this stuff was rejected by the governments without any rational reason until the research companies went either bankrupt or the patents expired so that research isn't profitable anymore. Since young hypogonadism is very rare, this probably won't change because almost noone cares or even knows about what happened. 

Long-term hCg is not adviced due to "limited data". We found that stuff almost 100 years ago. It's a joke, it's just not funny.

I've experience no atrophy of my testicles though supplementing for almost 20 years.  I remove my daily patch at night when the testicles are most active and apply what is left in the patch manually in the AM and then wear the new patch.

Yes, ABSOLUTELY!  Gonadotropin worked great when I got it over the internet when the internet was young and before oppressive regulation set-in.  

Of course, I got no help at all around 1960 during my ultra slow puberty and lagging T where it could have helped penis growth.  Supplementing at 50 of course did nothing for my penis size, but was great for my mental state, increasing my natural assertiveness and reducing anxiety.

I don't think I had actual hypogonadism, just abnormally low with delayed puberty.

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On 3/11/2020 at 11:32 AM, Klingsor said:

A much more reliable biological marker than stretching:

https://en.wikipedia.org/wiki/Digit_ratio

Yes, I'm too old to have been exposed to plastics, but my mother took amphetamines when pregnant which she had become addicted to during WWII when they were handed out like candy to servicemen.  My father had to stand watch for the Navy on a break water.  My mother took more dexadrine than he did.

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